This study has many high quality characteristics including high participation rates, prospective design, and objective data on TBI and dementia and a several medical confounders. It can be found here: Jesse R Fann et al. Lancet Psychiatry http://dx.doi.org/10.1016/S2215-0366(18)30065-8. The study includes 126,734 dementia cases in the analysis; 6,374 had experienced TBI. A potentially powerful study. The data clearly shows an increased risk of dementia diagnosis within the 2 years following TBI. However, there is no variation in risk between 4 and 14 years after TBI, suggesting a completely uniform acceleration, which would be hard to explain. There is also decreasing risk as age increases, yet older people are more vulnerable to dementia. This also is hard to explain. The authors call for more to be done to prevent TBI. Dementia is a growing problem. Motor and sports insurers would be especially sensitive to this issue if it turned out that TBI was a legal cause of dementia. Especiall

The judgment may be found here. The Court has decided that symptomless sensitisation is an injury. But there may have been some error. Background IgE sensitisation to platinum salts was detected by a routine skin prick test in five employees. Occupational exposure was the probable cause. The employer admitted breach of statutory H&S duty. Sensitisation is a necessary precursor to allergic reaction, but a person may remain free of symptoms for years and may never have an allergic reaction. What the Court seems not to have been advised though is that in a high proportion, sensitisation becomes undetectable if further contact with the allergen is avoided or remains below a certain level. There were no symptoms despite working in the same environment which had caused this biological change. It is implausible that all five became sensitised only moments before the test. More likley they were sensitised at some point between tests and had continued to be exposed to platinum salts until t